青云英语翻译

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调查效果的MIR214的静音在全国人大常委会的细胞诱导细胞自杀[CNE2,Sune1],caspase的活动检测到3。 结果表明,与LNA的控制,相对的活动的caspase3增加了[3±0.05]可折叠的CNE2单元格的LNA-antimir214组[p<0.001,图2b]。 同样,与LNA的控制,相对的活动,增加了caspase3±0.09[2]可折叠的,Sune1单元格的LNA-antimir214组[p<0.001,图2b

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调查的和平号空间站-214 沉默对细胞凋亡的全国人民代表大会 (CNE2 和 SUNE1),半胱氨酸蛋白酶-3 的活动影响被检测到。结果都表明,相比低噪声放大器控件,相对活性的半胱氨酸蛋白酶-3 增加了低噪声放大器-antimiR-214 组的 CNE2 细胞 (3.00±0.05) 倍 (P < 0.001,图 2B)。同样,与低噪声放大器控件相比,半胱氨酸蛋白酶-3 的相对活动增加由低噪声放大器-antimiR-214 组的 SUNE1 细胞 (2.73±0.09) 折叠 (P < 0.001,图 2B

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要调查沉默miR214的作用对全国人民代表大会细胞细胞凋亡(CNE2和SUNE1), Caspase-3的活动查出。结果显示,比较LNA控制, Caspase-3的相对活动由(3.00±0.05)在CNE2细胞的折叠增加了LNA-antimiR-214小组(P<0.001,图2B)。同样,比较LNA控制, (2.73±0.09)在LNA-antimiR-214小组(P<0.001,图2B SUNE1细胞的折叠增加的Caspase-3的相对活动

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